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A 1992 report associated the use of anabolic steroids with tinea versicolor, a fungal skin disease sensitive to sun exposure, in patients suffering from the disease.[3]) Another report related an association of tinea versicolor with the use of steroids.[4] In a case series of a young female athlete with tinea versicolor, the patient complained of persistent itching after steroid administration.[5]

A more recent case report includes an adolescent male (aged 14 years) presenting with frequent facial pain and tinea versicolor, who had recently been treated for multiple body aches using steroids.[6] Tinea versicolor occurs most commonly in association with steroid exposure, although it can also occur in association with a wide variety of medical conditions. Because of its prevalence, and because its diagnostic accuracy is not great, tinea versicolor might be confused with other systemic illnesses, such as a bacterial infection (eg, sinusitis) or sepsis.[7]

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There is not a great deal of information available on how tinea versicolor originated. The cause of the disease is uncertain. In some cases, the disease has been reported to be a congenital disease, in which the immune system is mistakenly attacked and produces an inflammatory reaction from the outer layer of skin.[5] Other causes are presumed to have occurred before the onset of tinea versicolor, such as environmental exposure to anaerobic conditions or excessive sunlight.[6] Evidence of a genetic cause has also been reported.[6] In general, there is no evidence that tinea versicolor is a bacterial infection, nor is there any evidence of a systemic infection. However, it is possible from the reports that it is a multifactorial disease.

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The cause of tinea versicolor is uncertain, and the pathophysiology remains unknown. Treatment is designed to minimize or eliminate discomfort and provide a safe environment. There is a lack of conclusive evidence that steroids cause any significant pathology or affect the normal flora of the body. A large volume of published research, in the form of case series and literature, has examined the pathophysiology of tinea versicolor. Only a few reports have examined the mechanisms by which tinea versicolor occurs. They include (1) exposure to sunlight, the main potential source of steroid exposure, although it should be remembered that this is far from certain when studying the pathophysiology in tinea versicolor; (2) inflammation and/or anemia,[4,5] caused by chronic or acute infections or inflammation of another organ; (3) an

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