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In a recent paper, Dr. D’Agostino and Dr. Boulay developed a mouse model that represents the typical human, albeit one with higher levels of anabolic hormones (and fewer anabolic compounds). A drug was administered to the mice and they were then tested on a number of measures of growth and/or strength, anabolic-androgenic steroids forms. Although the mice received high doses of anabolic hormones, the researchers found that, in comparison to control animals, the mice with higher levels of anabolic hormones displayed greater gains in size and strength. Dr, halo anabolic. D’Agostino commented that, “The key is that the mice developed greater increases in muscle size and strength when
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After careful review of the medical data, it has been hypothesized that declining levels rather than high levels of anabolic steroids are major contributors to prostate cancer (Prehn 1999)The primary objective of this study was to determine if the increased rate of prostate cancer observed in the study population (n = 1881) was a real increase in prostate cancer, or a real reduction in prostate cancer. The primary outcome was total prostate cancer and was calculated as the incidence of prostate cancer in men treated with placebo, 20 mg of DHT, or 40 mg of DHT. The secondary outcome was the incidence of total prostate cancer in men treated with either 10 or 20 mg of DHT once daily.
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The study design used in this study was a case-control study. There is much debate regarding the need for case-control studies in prostate cancer. A study was performed to establish that the incidence of primary and secondary prostate cancers were not related to the dose of the anabolic steroid in a double blind study. A total of 11,841 single- and dual-assay double-blind placebo-controlled studies were evaluated. Six trials met the selection criteria. For the primary analysis, the incidence of prostate cancer is calculated for each of these 6 trials.
Table 3 describes the primary study outcomes. The incidence of total prostate cancer was estimated, as is the incidence of multiple sites.
Overall Prostate Cancer Incidence No. of Prostate Cases Total Prostate Cases Primary Prostate Cancer Incidence No. of Prostate Cases Total Prostate Cases Primary Prostate Cancer Incidence No. of Prostate Cases Total Prostate Cases Primary Prostate Cancer Incidence n % n % n % n % n % n % n % n % n % n % 2,838 0 1,838 0 1,838 0 1,838 0 1,838 0 1,838 0 1,838 0 81.8 81.8 85.4 80.6 81.8 81.2 738 19 733 17 715 19 742 19 738 19 726 21 659 14 507 14 724 16 596 17 602 16 613 16 712 17 586 18 592 18 647 18 632 14 562 14 562 14 638 20 560 17 619 21 569 13 510 13 557 13 602 21 442 13 440
Source: (Hepburn, 2003)
Table 3: Prostate Cancer Incidence No. of Prostate Cases Total Prostate Cases Primary Prostate Cancer Incidence No. of Prostate Cases Total Prostate Cases Primary Prostate Cancer Incidence
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