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Trenbolone acetate vs Trenbolone Enanthate would be the same thing as comparing testosterone prop (a short ester) to testosterone enanthate (a longer acting ester)in regards to testosterone levels. The more effective esters would always lead you to more testosterone. However, if testosterone was actually derived directly from testosterone prop then this could change significantly. This is where you would see the “reproductive cycle” in action.

Reactions between the two types of esters may vary greatly:

The reason why Trenbolone is considered the best way to combat this problem is because there is a huge ratio of testosterone within all tissues within the body. To test whether your testosterone is elevated or down a certain level using Trenbolone is to test the level of your own TSH, which is the body’s natural hormonal signal. Trenbolone can give you a little extra boost, but will also raise your risks of hypogonadism where your TSH drops below 17.5. If your TSH is above 17.5 your thyroid is not producing enough thyroid hormone to normalise the levels on the TSH test to your target.

The other side of the coin, is that testosterone prop is used to treat people with a condition similar to prostate cancer. And, as a result, it can lower TSH levels which can further impact your Tert’s prognosis. If you have prostate cancer, Trenbolone might actually help. We are not talking about a miracle serum. Rather, it’s important to note that while the hormone can reduce prolactin levels (a signal that your body is in pre-menstrual), the hormone cannot prevent it. Trenbolone has an effect, but it’s only about half as strong as it first appears. Even if you know you have prostate cancer you may still be concerned how strong Trenbolone will actually be in the absence of other treatments. It’s important to note that Trenbolone also does not affect any other hormone that we use (i.e. it is not like dihydrotestosterone or testosterone progesterone).

This is all part of an explanation for why Trenbolone is now being prescribed with all sorts of other medications and therapies to help manage low T levels. With this kind of prognosis and treatment options coming in it’s not surprising people are looking to get back into their “normal” range, and taking as much T as possible.

With the advent of synthetic testosterone the amount of T that is created has gone up drastically, from about 11% to 15% in the last few years. The reason being is because of the extra

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